Ozi ndị ọzọ ― Igbo

Otu nwaanyị dị afọ iri abụọ na isii bịara ụlọọgwụ anyị na-eme mkpesa maka ọkụ ọkụ n'ụkwụ ya na-egbu ya nke ọ na-enwe kemgbe afọ iri. N'agbanyeghị iji ude steroid na ude tazarotene, ihe ọkụ ọkụ ahụ adịchaghị mma. O nwechaghị akụkọ ezinụlọ metụtara ya. Mgbe anyị lere ya anya, anyị hụrụ n'ihu ụkwụ ya na-esichị ike, bụ́ nke dabara n'ọrịa a na-akpọ lichen amyloidosis.
A 26-year-old woman presented to our clinic with an itchy rash on her legs that had persisted for 10 years. The rash had previously been treated with topical steroids and tazarotene cream, with no improvement. The patient’s family history was noncontributory. A physical examination showed discrete and coalescing hyperkeratotic tan-brown papules on the pretibial surfaces, consistent with lichen amyloidosis.

Lichen Amyloidosis bụ ọnọdụ akpụkpọ ahụ na-adịghị ahụkebe jikọtara ya na itching na-adịgide adịgide nke amaghị ihe kpatara ya. Ọ na-apụtakarị dị ka ụyọkọ gbalitere agba agba na elu akpụkpọ ahụ. Lichen Amyloidosis na-apụtakarị na ndị gbara afọ 50 ruo 60 ma ọ dị mwute na ọ nweghị ọgwụgwọ maka ya. Ọgwụgwọ dị ugbu a anaghị arụkarị ọrụ nke ọma.
Lichen Amyloidosis (LA) is an uncommon, primary cutaneous amyloidosis associated with chronic, idiopathic pruritus. Clinical presentation of LA includes skin colored to hyperpigmented, papules coalescing into plaques with a rippled appearance on the extensors.1 LA most commonly presents in the fifth to sixth decade of life and has no curative treatments. Overall response to current therapies is poor.

Lichen amyloidosis bụ ọnọdụ akpụkpọ ahụ na-egbu mgbu na-adịte aka. Amara ya maka ụyọkọ nke mkpọmkpọ ebe a na-ahụkarị nke a na-ahụ n'azụ, ntan, apata ụkwụ, na ogwe aka. Mgbe a na-enyocha ya n'okpuru microscope, Lichen amyloidosis na-egosi njupụta nke amyloid n'ime elu akpụkpọ ahụ yana ịgbatị na mmụba nke oyi akwa akpụkpọ ahụ. Ọ bụ ezie na a ghọtabeghị ihe kpatara Lichen amyloidosis nke ọma, nnyocha ndị gara aga ejikọtawo ya na ihe ndị dị ka ikpochapụ ma ọ bụ esemokwu na akpụkpọ ahụ, ọnwụ cell, ọrịa nje, na ndị ọzọ. Lichen amyloidosis yiri ka ejikọtara ya na ọtụtụ ọnọdụ akpụkpọ anụ ndị ọzọ (atopic dermatitis, lichen planus, mycosis fungoides).
Lichen amyloidosis (LA) is a chronic pruritic skin disorder characterized by multiple grouped hyperkeratotic papules, predominantly located on the back, shins, thighs, and arms. Histological analysis of LA shows amyloid deposition in the papillary dermis and hyperkeratosis and acanthosis of the epidermis. The exact pathogenesis of LA has not yet been elucidated; however, prior reports have implicated frictional epidermal damage, apoptosis, viral infection, and many other triggers. LA is reportedly associated with several skin disorders, including atopic dermatitis (AD), lichen planus, and mycosis fungoides.